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In the APP (Swe)/[PS1(L166P)] transgenic (Tg) model of AD, germ-free status reduced cerebral A load compared to conventionally raised APP rodents (Harach et al., 2017), implicating microbiota as a direct or indirect modifier of peripheral amyloidosis (discussed later in Section Gut Microbiome-Mediated Fibril Formation)

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Research in animal models indicates that elevated NNMT expression is associated with reduced levels of the glucose transporter GLUT4

Beyond these direct actions, emerging insights indicate that BPC-157 may also influence gene expression